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Likewise, a study utilizing FDG-PET imaging of brain glucose metabolism revealed that PD is characterized by hypermetabolism in the putamen, pallidum, thalamic sensorimotor cortex, pons, and cerebellum, along with relative hypometabolism in the posterior temporal-parietal lobe, occipital lobe, and occasionally the frontal lobe, particularly in PD patients with cognitive impairment [36]

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When the riboflavin lack was corrected, some of the early morphologic and cell motility alterations in the gastrointestinal tract were observed in weanling rats given a riboflavin-deficient diet until weaning was irreversible (109, 110)

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