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Unlike most peptides that are produced in glands or organs and travel to target tissues, MOTS-c originates inside the cell and acts as a local and systemic signal of mitochondrial stress and energy status

Our results also suggest that folate and cobalamin, and to a lesser extent, vitamin B6, become limiting factors in tHcy regulation when riboflavin status is replete and support previous reports of crosstalk between these vitamins affecting homocysteine regulation

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