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This section synthesizes the mechanisms by which current therapeutic interventions (including EPO and its analogs, nitric oxide (NO) donors, and antioxidants) counteract accelerated eryptosis, thereby extending RBC survival and alleviating renal anemia, while also discussing their clinical utility, limitations, and relevance across CKD stages

Simultaneously, its glucagon receptor agonism supports hepatic glucose output (gluconeogenesis), helping maintain blood glucose within a stable range

GLP-1 receptor agonists such as semaglutide are mostly safe

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