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163 At the lesion core therefore, all neural cells die by necrosis, which leads to a massive release of damage-associated molecular patterns (DAMPs), including release of glutamate and ATP, which propagate excitotoxicity

placebo Rapid onset of activity, with separation from placebo observed as early as Day 8 HAM-D17 response rate reached 71.4% at Day 22 in the 20 mg arm Favorable safety profile with no SAEs, no Grade 3+ treatment-related AEs, and no discontinuations due to adverse events KH607 is designed with preferential selectivity for the 532 GABAA receptor subtype, which may offer a differentiated approach within the neuroactive steroid/GABAA PAM landscape