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Future research should more clearly define the cognitive constructs being trained, align intervention designs with established cognitive models, and employ standardized neuropsychological measures to assess domain-specific effects

In particular, the E4 allele mediated decline in MCA and LCAs within the cerebrovasculature at 50 weeks may indicate that cerebrovascular mitochondrial FAO flux is impaired earlier in E4 relative to other alleles, especially in female E4s who fail to increase acylcarnitines with age, and at older ages in the same animal model have been shown to fail to increase AA as an alternative energy source compared to male E4s ( APOE had an impact on the production of L-carnitine metabolites associated with vascular dysfunction

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