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In chronic CCl4/HFD-induced liver injury, HSYA ameliorated serum biochemical indicator, reduced mRNA expression levels of pro-fibrotic factors and elevated hepatoprotective factors, which revealing that PPAR- and p38 MAPK signaling play pivotal roles in the prevention of liver fibrosis (Liu et al., 2014)

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92 , 128 These effects translate into reduced synaptic loss and improved cognitive performance, suggesting that incretin signalling contributes to neuronal survival through both antiinflammatory and bioenergetic pathways

Gut microbiota in human metabolic health and disease