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Oxidative stress: a common denominator in the pathogenesis of amyotrophic lateral sclerosis

However, for analyses involving non-normally distributed ACC GABA levels in controls, Spearmans rho was employed

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Preclinical data indicate modulation of inflammatory and pro-fibrotic signalling pathways relevant to lung fibrosis, yet direct demonstration of reduced fibroblast activation or collagen deposition in established pulmonary fibrosis models is currently lacking