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The mechanistic plausibility is clear: if a compound supports angiogenesis and collagen synthesis in wound environments, the theoretical rationale for faster tissue repair is sound
Neuroscience (1988) 27:77783.10.1016/0306-4522(88)90182-0 71 KramrEAArmstrongDLIkedaSWaynerMJHardingJWWrightJW
Biologic therapies, mainly including anti- tumor necrosis factor-alpha (TNF-) monoclonal antibodies, anti-integrin monoclonal antibodies, interleukins (IL)-12/IL-23 antagonists and others, demonstrated efficacy in managing moderate to severe UC [12]
Other studies have shown that ROS production in response to glutamine deprivation increases the expression of p53-dependent genes ( Gadd45a, Cdkn1, and Sestrin2 ) via B55 or IKK 124,125
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