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Research demonstrates that it promotes synapse formation at approximately 10 million times the potency of BDNF itself, working through the hepatocyte growth factor (HGF) and c-Met receptor pathway

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In these vessels, occlusion syndrome and occlusion-like syndrome development and BPC 157 therapy, and the activation of the collateral pathways to mobilize trapped blood volume via the azygos vein was shown to play a particular role as a rescuing pathway [14,15,16,17,18,19,20,21,22,23]