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Clinical data shows all three sites deliver equivalent total drug exposure over the course of a week

Inhibition of HDAC enhances STAT acetylation, blocks NF-B, and suppresses the renal inflammation and fibrosis in Npr1 haplotype male mice

Khavinson VK, Anisimov VN

This provides direct cellular and molecular evidence for understanding the mechanisms of ferroptosis imbalance in AKD and CKD pathogenesis while offering crucial theoretical and data support for subsequent intervention strategies targeting ferroptosis-promoting pathways in kidney diseases (e.g., cell type-specific iron metabolism modulation and lipid peroxidation inhibition)