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In contrast, cuproptosis represents a separate copper-dependent pathway in which excess copper directly targets mitochondrial metabolism, inducing cell death through aggregated lipoylated proteins and iron-sulfur (Fe-S) cluster loss, independent of apoptotic signaling [3, 8]

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Exogenous glycine betaine treatment enhances chilling tolerance of peach fruit during cold storage

1,2 It is now evident that other factors, including defective actions of the gastrointestinal (GI) incretin hormones glucagon-like peptide1 (GLP-1) and glucose-dependent insulinotropic polypeptide (GIP), also play significant roles