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The answer seems to lie in the brains reward system, and specifically, a chemical called dopamine

Results: Treatment of TAC model mice with PQQ significantly suppressed myocardial hypertrophy and fibrosis, in addition to inhibiting the ferroptotic death of hypertrophic myocardial cells in vivo

In this post I compare PNGs DSIP volumes to CDFs globally, and describe trends in DSIP expenditure

The GLP-1 receptor is expressed in all these subcortical target regions and also in cortical and hippocampal regions that do not get axonal input from the GLP-1 neurons in the hindbrain (Randolph et al., 2024)