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Tailoring a molecule to this specific purpose requires a substantial understanding of how the current class of GLP-1R agonists access and interact with the satiating and aversive neuronal receptor populations

131,132,133 MSCs have been shown to directly degrade Keap1 and activate Nrf2 activity, which increases the levels of HO-1 and NQO-1, inhibits the infiltration of colonic inflammatory cells and the expression of proinflammatory cytokines, and maintains the permeability of the mucosal barrier of colon epithelial cells

References: Enteroendocrine L Cells Sense LPS After Gut Barrier Injury to Enhance GLP-1 Secretion