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While a fibrotic matrix initially contributes to tissue repair, after injury, chronic and persistent kidney damage in the development of CKD leads to uncontrolled fibrotic matrix deposition, resulting in reduced blood supply, destruction of normal organ structure, and eventual glomerulosclerosis, renal tubular atrophy, and interstitial fibrosis [9, 10]

All study participants were recommended to limit high-carbohydrate and high-fat foods and increase their physical activity levels as standard treatment for NAFLD 12,13

aligns with phase 2 obesity trial readouts

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