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As it was very important to increase the intravenous half-life of GLP-1 beyond that which could be obtained with dipeptidyl peptidase IV (DPP-IV) stabilization (36), fatty-acid derivatization was employed

This mechanism, in part, explains the lag in muscle glucose uptake following appearance of insulin in the blood

Since GM has been shown to improve cellular and molecular functions via mitigating TBI-induced pathologies such as BBB permeability, neuroinflammation, astroglia activation, and mitochondrial dysfunction, herein we discuss how a dysbiotic gut environment, which in fact, contributes to central nervous system (CNS) disorders during brain injury and how to potentially ward off these harmful effects

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