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Tissue repair research sometimes pairs IGF-1 LR3 with BPC-157 and TB-500 to examine whether IGF1R-driven anabolism combined with angiogenesis (BPC-157) and cellular migration (TB-500) accelerates repair beyond what either mechanism produces alone
Cellular NAD+ levels decline with age and metabolic stress, which has prompted interest in raising them through precursors or direct administration for energy, metabolic and longevity goals [11]
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