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Other mechanisms like mutations that enhance cytosolic levels or intensify intracellular signaling have also been associated with ASD (, andincreased levels could activate an immune response similar to that detected in ASD brains ( Mitochondrial dysfunction in ASD is further supported by the presence of non-specific biomarkers including reduction in carnitine and increases in lactate, pyruvate, creatine kinase, aspartate aminotransferase and alanine aminotransferase compared to control groups (Rossignol and Frye, 2012)

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pagei was able to prevent/attenuate the development of adiposity, dyslipidemia, hypertension and hyperglycemia observed in a Metabolic Syndrome (MS) model induced by chronic administration of a high-sucrose diet (HSD) to male rats