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Dysbiosis-driven disruptions of the MGBA have been implicated in both the development of common stroke risk factors, such as obesity, diabetes, and atherosclerosis, and in poorer recovery post-stroke, with age-related changes further compounding MGBA dysfunction (Honarpisheh et al., 2022)

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We also report increased oxidative DNA damage in our BWS hepatocytes, in conjunction with the upregulation of the OGG1 gene

Acting on insulin and glucagon levels, incretins, and thus TZP, may have indirect effects also on the liver and muscle