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The findings are not surprising, but they help to answer an important, unanswered question about GLP-1 drugs and sexual health

Whilst mTORC1 activates S6 kinase, which phosphorylates serine residues on IRS which in turn uncouples IRS from the receptor and its substrates, mTORC2 can reactivate this signalling

Yoshida, K., Sakai, O., Honda, T., Kikuya, T., Takeda, R., Sawabe, A., Inaba, M., & Koike, C

In PNA mice, GLP1/E therapy caused upregulation of pathways involved in the metabolism of non-coding RNA, estrogen-dependent gene expression, autophagy, vesicle-mediated transport, and signaling by nuclear receptors, whereas proteins related to inflammation (ROS and RNS production in phagocytes), cellular response to starvation, signaling by insulin receptor, apoptosis, and immune system were downregulated