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Homocysteine can then either be remethylated back to methionine by using a methyl group which is delivered by methyltetrahydrofolate or it is further converted into cystathionine by cystathionine b-synthase (CBS)
Inactivation of DHODH in cancer cells with low GPX4 expression significantly increased intracellular lipid peroxidation, while in cancer cells with high GPX4 expression, DHODH inactivation synergized with inducers to enhance ferroptosis
T.MortazaviS
Prospects In recent years, numerous studies have commenced exploring the interplay between ferroptosis, immune cells, and autoimmune diseases, accentuating the dual role of ferroptosis in pathology
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