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In primary neurons under excessive stimulation by NMDARs, kinase D-interacting substrate of 220 kDa exon 33 (Kidins220-C33) was downregulated by protease calpain activation and was mainly expressed in neurons

In This Review Cancer therapy based on cell death induction has become an important research topic, and ferroptosis, a newly discovered form of cell death, has gained general attention

Finally, it is important to note that this review is a synthesis of the existing literature and, as such, does not involve the collection or analysis of original data

Orian Shirihai, Ph.D., Associate Professor at Boston University School of Medicine, and Marc Liesa, Ph.D., a research associate on Shirihais team at Boston University School of Medicine.