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Here, we report that the combination therapy of As 2 O 3 with L-buthionine-sulfoximine (BSO), which inhibits a critical step in glutathione synthesis, effectively enhanced in vitro growth inhibition effect of As 2 O 3 on all 11 investigated cell lines arising from prostate, breast, lung, colon, cervix, bladder, and kidney cancers, compared with As 2 O 3 treatment alone

Our findings underscore the critical role of the ECM in orchestrating MC function and differentiation, highlighting the need for further studies to fully understand the intricate crosstalk between mechanical parameters and ECM ligands in the cellular microenvironment (Fig