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Intramuscular B12 injections represent the standard treatment for deficiency due to malabsorption (such as pernicious anaemia) or when neurological symptoms are present

Under hyperglycemia, lipids, proteins, and nuclear acids can form advanced glycation end products, interact with the corresponding receptor and finally trigger the activation of inflammation, oxidative stress, and mitochondrial dysfunction, which ultimately induces neuronal injury ( 3.1 In vitro evidence Qi et al

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Recommendation 4.4 The minimum dose of NTBC should be used to achieve a blood concentration of 4060 mol/L and/or blood SA level within the normal range of the reference laboratory