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In primate females, and in human females in particular, an adaptative mechanism during stress is represented by the reduction of reproductive axis activity, blocking a function which is not really essential to survive

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Since the biological features of FPLD2 are related to metabolic alterations including insulin resistance, mitochondrial dysfunction and a reactive oxygen species (ROS) increase in the cytoplasm 11 we have examined the metabolic and stress connections of the specific DEGs associated with each C2C12 cluster