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As summarized in Journal Watch Psychiatry, elevated c reactive protein levels (increased inflammation) in patients with untreated depression was associated with higher levels of glutamate in the left basal ganglia

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GLP-1RAs mitigate these adverse impacts by enhancing fatty acid oxidation via the Creb5/NR4a1 signaling axis, thereby reducing mitochondrial damage, lipid accumulation, and ATP deficiency (77, 78, 83)

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