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Abstract Objectives The purpose of this study was to examine the effect of cigarette smoking on plasma uric acid concentration and to determine the correlation between this parameter and the biological tobacco markers, plasma thiocyanate and urinary cotinine

We found that inhibiting the expression or function of SREBP2 and HMGCR reduced intracellular cholesterol levels (Figure 3CF), thereby inhibiting CSE-induced secretion of inflammatory factors IL6 and IL8 in airway epithelial cells (Figure 3D, E and G)

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